Accelerated biological aging and the dynamic transitions of stroke and dementia: A multi-state trajectory analysis.

Wenke Cheng, Liuyu Chen, Pinfang Kang, Bi Tang, Shuijing Zhang, Guangji Zhang, Zhongyan Du, Yuli Huang

Department of Cardiology, The First Affiliated Hospital of Bengbu Medical University, Bengbu 233000, China; Zhejiang Key Laboratory of Blood-Stasis-Toxin Syndrome, Zhejiang Chinese Medical University, Hangzhou 310053, China.Department of Cardiology, The First Affiliated Hospital of Bengbu Medical University, Bengbu 233000, China; Zhejiang Key Laboratory of Blood-Stasis-Toxin Syndrome, Zhejiang Chinese Medical University, Hangzhou 310053, China. · pubmed

Paper

Abstract

Accelerated biological aging has been implicated in increased vulnerability to age-related diseases. However, its role in neurovascular disease trajectories remains unclear. We aimed to investigate how biological age acceleration (BAA), estimated by two established aging algorithms, influences the sequential development of stroke, dementia, comorbidity accumulation, and death.

The following analysis is generated by AI and may contain errors. It is separate from the authors’ abstract and is not peer review.

Research summary

The paper claims that biological age acceleration influences the sequential development of stroke, dementia, comorbidity accumulation, and death. This research is relevant as it explores the relationship between biological aging and the progression of age-related diseases, potentially contributing to understanding the mechanisms of aging and its impact on longevity.

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