Melatonin mitigates ovarian aging through regulation of the YTHDF2/m
Abstract
<p indent="0mm">Ovarian aging is a natural process characterized by a decline in both the quantity and quality of oocytes, which subsequently leads to diminished fertility, particularly in women over the age of 35. Given the societal trend toward postponing childbirth, it is imperative to understand the molecular mechanisms that underpin ovarian aging to address infertility issues. Melatonin (MT) is recognized for its therapeutic potential in mitigating ovarian aging; however, the specific epigenetic mechanisms involved, particularly concerning m
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Research summary
Melatonin mitigates ovarian aging through the regulation of the YTHDF2/m pathway. The paper addresses the molecular mechanisms of ovarian aging, which is directly related to understanding and potentially mitigating aspects of aging and fertility decline.
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