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RESEARCH PAPER / 2026-02-12

Characterizing the SASP-Dependent Paracrine Spreading of Senescence Between Human Brain Cell Types

Russo, T., Riessland, M.

Stony Brook UniversityStony Brook University · biorxiv

Abstract

One of the defining phenotypes of a senescent cell is the senescence-associated secretory phenotype (SASP), which can propagate senescence in neighboring cells both in vitro and in vivo. Importantly, this paracrine spreading of senescence can act in a cell non-autonomous manner, influencing neighboring cell populations and contributing to immune cell recruitment. As cellular senescence has recently been linked to both age-related neurodegenerative phenotypes and local inflammation and is more clearly defined across brain cell types in a cell-type-dependent manner, an urgent question remains regarding how a cell-type-specific paracrine spreading of senescence occurs in the brain. Here, we set out to profile the cell-type-specific features of the SASP and characterize the directionality of paracrine senescence-spreading between major brain cell types. Through this analysis, we identified key SASP ligand-receptor pairs involved in this paracrine dissemination. Targeting these factors with specific inhibitors, we prevented the paracrine spreading of senescence in a brain cell-type-dependent manner. Taken together, we identified specific SASP targets for therapeutic intervention in the context of human brain cells and thereby informed the SASP-dependent reaction of immune cells and age-related tissue dysfunction across both normal aging and models of neurodegenerative disease.

The following analysis is generated by AI and may contain errors. It is separate from the authors’ abstract and is not peer review.

Research summary

The paper identifies specific SASP ligand-receptor pairs that facilitate paracrine senescence spreading in brain cell types. This research is relevant as it addresses the mechanisms of cellular senescence, which is a contributing factor to aging and age-related diseases, potentially leading to therapeutic interventions that target the root causes of these conditions.

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